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However, the patient revealed a nasal obstruction and chronic mouth breathing condition, which induced an altered maxillary arch development with a high narrow vault. Despite the fact that non-inflammatory destructive periodontal disease has been reported in the literature, its etiology, progression and treatment, are still indefinite and therefore, are open to discussion [ 20 , 21 ]. The patient presented a satisfactory oral hygiene, no apparent detectable bacterial plaque, calculus, and periodontal inflammation Fig. The patient also had anxiety and constant stress development because of his work, and realized that several times, an unconscious centric bruxism occurred during the daytime and, that after sleeping time, he felt pain around his temporo-mandibular joint, mainly, when he was under stress.

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The premature contact deflected the mandible forward and away from the centric relation, to the maximum intercuspation causing an effective and deleterious contact in the anterior teeth. The posterior premature contact was, adjusted in the centric relation, in order to stabilize the mandible, and to eliminate a deleterious contact in the anterior teeth. However, the patient revealed a nasal obstruction and chronic mouth breathing condition, which induced an altered maxillary arch development with a high narrow vault.

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Despite the fact that non-inflammatory destructive periodontal disease has been reported in the literature, its etiology, progression and treatment, are still indefinite and therefore, are open to discussion [ 20 , 21 ]. In majority of the patients suffering from periodontal disease, the consequence is the development of periodontal pocket [ 13 , 19 ]. In this paper, we presented a case that demonstrated the features of the disease and discussed the probable etiology and possible therapy. The patient also had anxiety and constant stress development because of his work, and realized that several times, an unconscious centric bruxism occurred during the daytime and, that after sleeping time, he felt pain around his temporo-mandibular joint, mainly, when he was under stress.

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No eccentric movements were defective. The inflammatory process inactivates the bacteria, but produces the liberation of bacterial and neutrophils products such as enzymes, which induce periodontal tissue destruction by lytic activities [ 12 , 13 , 16 - 18 ]. The patient also submitted to standard protocols, for the evaluation of the hematological and endocrine markers, as used by Repeke et al.

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An NIDPD case was studied in order to demonstrate features of the disease, and discuss the possible etiology and treatment. Non-Inflammatory Destructive Periodontal Disease NIDPD , is a severe destructive periodontal disease, which is characterized by periodontal attachment loss, alveolar bone loss, generalized gingival recession without pathognomonic sign of inflammation, and periodontal pocket development [ 20 , 21 ]. Conventional periodontal therapy and antimicrobial therapy are ineffective, in preventing further progression of the disease [ 20 ]. In the anamnesis questionnaire, the patient informed that there was no previous specific periodontal treatment, neither was he taking any medication for at least 3 months prior to this study, and denied any pre-existing systemic conditions or significant history of harmful habits.

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The inflammatory process inactivates the bacteria, but produces the liberation of bacterial and neutrophils products such as enzymes, which induce periodontal tissue destruction by lytic activities [ 12 , 13 , 16 - 18 ]. Therefore, the characteristics of the most common periodontal disease are: In majority of the patients suffering from periodontal disease, the consequence is the development of periodontal pocket [ 13 , 19 ].

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The patient presented a satisfactory oral hygiene, no apparent detectable bacterial plaque, calculus, and periodontal inflammation Fig. Non-Inflammatory Destructive Periodontal Disease NIDPD , is a severe destructive periodontal disease, which is characterized by periodontal attachment loss, alveolar bone loss, generalized gingival recession without pathognomonic sign of inflammation, and periodontal pocket development [ 20 , 21 ]. Despite the fact that non-inflammatory destructive periodontal disease has been reported in the literature, its etiology, progression and treatment, are still indefinite and therefore, are open to discussion [ 20 , 21 ].

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An NIDPD case was studied in order to demonstrate features of the disease, and discuss the possible etiology and treatment. Concerning the dental occlusion conditions, a significant deflective premature contact in the centric relation was verified. Therefore, the characteristics of the most common periodontal disease are:

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4 Commentsto Fumico

  1. Nikom says:

    Conventional periodontal therapy and antimicrobial therapy are ineffective, in preventing further progression of the disease [ 20 ]. This is an open access article licensed under the terms of the Creative Commons Attribution-Non-Commercial 4.

  2. Malashicage says:

    Conventional periodontal therapy and antimicrobial therapy are ineffective, in preventing further progression of the disease [ 20 ].

  3. Vudozil says:

    The inflammatory process inactivates the bacteria, but produces the liberation of bacterial and neutrophils products such as enzymes, which induce periodontal tissue destruction by lytic activities [ 12 , 13 , 16 - 18 ]. Non-Inflammatory Destructive Periodontal Disease NIDPD , is a severe destructive periodontal disease, which is characterized by periodontal attachment loss, alveolar bone loss, generalized gingival recession without pathognomonic sign of inflammation, and periodontal pocket development [ 20 , 21 ].

  4. Dibei says:

    Conventional periodontal therapy and antimicrobial therapy are ineffective, in preventing further progression of the disease [ 20 ]. In this paper, we presented a case that demonstrated the features of the disease and discussed the probable etiology and possible therapy.

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